Heart Failure with Preserved Ejection Fraction Induces Molecular, Mitochondrial, Histological, and Functional Alterations in Rat Respiratory and Limb Skeletal Muscle
European Journal of Heart Failure2015Vol. 17(3), pp. 263–272
Citations Over TimeTop 1% of 2015 papers
T. Scott Bowen, Natale Rolim, Tina Fischer, Fredrik H Bækkerud, Alessandra Medeiros, Sarah Werner, Eivind Brønstad, Øivind Rognmo, Norman Mangner, Axel Linke, Gerhard Schüler, Gustavo José Justo da Silva, Ulrik Wisløff, Volker Adams
Abstract
Our findings are the first to demonstrate that HFpEF induces significant molecular, mitochondrial, histological, and functional alterations in the diaphragm and soleus, which were attenuated by exercise training. These data therefore reveal novel mechanisms and potential therapeutic treatments of exercise intolerance in HFpEF.
Related Papers
- → Sarcopenic Obesity and the Pathogenesis of Exercise Intolerance in Heart Failure with Preserved Ejection Fraction(2015)76 cited
- → Exercise Intolerance in Heart Failure with Preserved Ejection Fraction(2021)26 cited
- → Skeletal muscle abnormalities in heart failure with preserved ejection fraction(2022)20 cited
- → Effects of Exercise Training on Cardiac Function in Heart Failure with Preserved Ejection Fraction(2020)13 cited
- → Heart Failure with Preserved Ejection Fraction: Pathogenesis, Diagnosis, Exercise, and Medical Therapies(2022)7 cited