Cross‐resistance, inheritance and biochemical mechanisms of imidacloprid resistance in B‐biotype Bemisia tabaci
Pest Management Science2009Vol. 65(11), pp. 1189–1194
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Abstract
Although oxidative detoxification mediated by P450 monooxygenases is involved in imidacloprid resistance in the NJ-Imi strain of B-type B. tabaci, target-site modification as an additional resistance mechanism cannot be ruled out. Considering the high risk of cross-resistance, neonicotinoids should be regarded as a single group when implementing an insecticide rotation scheme in B. tabaci control.
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