Neural Mechanisms Underlying Hyperphagia in Prader‐Willi Syndrome
Obesity2006Vol. 14(6), pp. 1028–1037
Citations Over TimeTop 10% of 2006 papers
Laura M. Holsen, Jennifer R. Zarcone, William M. Brooks, Merlin G. Butler, Travis I. Thompson, Jasjit S. Ahluwalia, Nicole L. Nollen, Cary R. Savage
Abstract
Results point to distinct neural mechanisms associated with hyperphagia in PWS. After eating a meal, the PWS group showed hyperfunction in limbic and paralimbic regions that drive eating behavior (e.g., the amygdala) and in regions that suppress food intake (e.g., the medial PFC).
Related Papers
- → Emotion, olfaction, and the human amygdala: Amygdala activation during aversive olfactory stimulation(1997)804 cited
- → Distinct Roles of Rodent Orbitofrontal and Medial Prefrontal Cortex in Decision Making(2010)392 cited
- → Different Time Courses for Learning-Related Changes in Amygdala and Orbitofrontal Cortex(2011)127 cited
- → Inhibitory Control and Affective Processing in the Prefrontal Cortex: Neuropsychological Studies in the Common Marmoset(2000)212 cited
- → Faculty Opinions recommendation of Distinct roles of rodent orbitofrontal and medial prefrontal cortex in decision making.(2010)