Atypical Hemolytic–Uremic Syndrome
New England Journal of Medicine2009Vol. 361(17), pp. 1676–1687
Citations Over TimeTop 1% of 2009 papers
Abstract
The hemolytic–uremic syndrome, which is characterized by nonimmune hemolytic anemia, thrombocytopenia, and renal impairment, occurs most frequently in young children. Most cases are secondary to infection with Escherichia coli O157:H7 and other Shiga-toxin–producing strains. However, approximately 10% of cases are atypical and not associated with infection. This article reviews current concepts about the pathobiology of atypical hemolytic–uremic syndrome and its diagnosis and management.
Related Papers
- → A Putative Microcin Amplifies Shiga Toxin 2a Production of Escherichia coli O157:H7(2019)15 cited
- → Potential for using antibiotics combined with a Shiga toxin-absorbing agent for treating 0157:H7 Escherichia coli infections(2002)2 cited
- Effect of Mitomycin C on the Expression of Shiga Toxin from Escherichia coli O157(2008)
- Isolation and PCR identification of Salmonella and Shiga Toxin-producing Escherichia coli from Tibetan Sheep(2013)
- → A putative microcin amplifies Shiga toxin 2a production of Escherichia coli O157:H7(2019)