JTE‐013 supplementation improves erectile dysfunction in rats with streptozotocin‐induced type Ⅰ diabetes through the inhibition of the rho‐kinase pathway, fibrosis, and apoptosis
Andrology2019Vol. 8(2), pp. 497–508
Citations Over TimeTop 10% of 2019 papers
Kang Liu, Kai Cui, Hao Feng, Ran Li, Lin Huang, Yinwei Chen, Y. Zhang, Zhijian Chen, Hang Yuan, M. Li, Tai Wang, Ruzhu Lan, J. Liu, Ke Rao, Bo Wen
Abstract
JTE-013 supplementation partially improved erectile function in rats with DMED, likely by inhibiting smooth muscle contraction, corporal fibrosis, and apoptosis.
Related Papers
- → RhoA and Rho Kinase Activation in Human Pulmonary Hypertension: Role of 5-HT Signaling(2009)167 cited
- → A possible role of RhoA/Rho-kinase in experimental spinal cord injury in rat(2002)111 cited
- → Endothelin-1 Upregulates Activin Receptor-Like Kinase-1 Expression via Gi/RhoA/Sp-1/Rho Kinase Pathways in Human Pulmonary Arterial Endothelial Cells(2021)8 cited
- → RhoA and Rho kinase activity is increased in scleroderma microvascular smooth muscle cells(2007)
- → Abstract 1633: RhoA and Rho Kinase Activation in Human Pulmonary Hypertension - Role of 5-HT Signaling(2008)