Sodium tanshinone IIA silate inhibits oxygen‐glucose deprivation/recovery‐induced cardiomyocyte apoptosis via suppression of the NF‐κB/TNF‐α pathway
British Journal of Pharmacology2013Vol. 169(5), pp. 1058–1071
Citations Over TimeTop 10% of 2013 papers
Abstract
The inhibitory effects of STS on TNF-α and positive feedback signalling of the NF-κB/TNF-α pathways may play important roles in myocardial protection against ischaemia/reperfusion. These protective effects of STS are mediated by suppressing JNK activity through activation of the PI3K-Akt pathway.
Related Papers
- Effect of tanshinone IIA on TLR4 /NF-κB signal pathway of endothelial cells(2011)
- → [Hypoxia upregulates the expression of annexin A1 in lung adenocarcinoma a549 cells].(2012)
- Expression of cyclooxygenase-2 in IL-1β-stimulated mesangial cells is medicated by NF-κB/IκB signal pathway(2004)
- Time-dependent changes of white blood cell apoptosis under room temperature conservancy and its molecular mechanism(2003)
- → Affinity interactions of Actin with HS1 and Annexin I, II, IV, V and VI in RAW264.7 macrophages in vitro and in the Fc-receptor complex(2021)