The low‐expression programming of 11β‐HSD2 mediates osteoporosis susceptibility induced by prenatal caffeine exposure in male offspring rats
British Journal of Pharmacology2020Vol. 177(20), pp. 4683–4700
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Hao Xiao, Zhixin Wu, Bin Li, Yangfan Shangguan, Jean‐François Stoltz, Jacques Magdalou, Liaobin Chen, Hui Wang
Abstract
PCE caused osteoporosis susceptibility in male adult offspring, which attributed to the low-functional programming of 11β-HSD2 induced by corticosterone via GR/HDAC11 signalling.
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