Intranasal insulin improves mitochondrial function and attenuates motor deficits in a rat 6‐OHDA model of Parkinson's disease
CNS Neuroscience & Therapeutics2021Vol. 27(3), pp. 308–319
Citations Over TimeTop 10% of 2021 papers
Farideh Iravanpour, Leila Dargahi, Mohsen Rezaei, Masoud Haghani, Reza Heidari, Neda Valian, Abolhassan Ahmadiani
Abstract
Considering the prominent role of mitochondrial dysfunction in PD pathology, IN insulin as a disease-modifying therapy for PD should be considered for extensive research.
Related Papers
- → Ischaemic Preconditioning Inhibits Opening of Mitochondrial Permeability Transition Pores in the Reperfused Rat Heart(2003)342 cited
- → Preconditioning and postconditioning: The essential role of the mitochondrial permeability transition pore(2007)260 cited
- → Mitochondrial permeability transition pore-dependent necrosis(2022)148 cited
- → Aging and Calorie Restriction Oppositely Affect Mitochondrial Biogenesis through TFAM Binding at Both Origins of Mitochondrial DNA Replication in Rat Liver(2013)79 cited
- → Sanglifehrin A inhibits opening of mitochondrial permeability transition pore (MPTP) during ischemia/reperfusion in the adult but not in the immature heart(2006)