Lipoprotein (a) and Low‐density lipoprotein apolipoprotein B metabolism following apheresis in patients with elevated lipoprotein(a) and coronary artery disease
European Journal of Clinical Investigation2018Vol. 49(2), pp. e13053–e13053
Citations Over TimeTop 14% of 2018 papers
Louis Ma, Elisa Waldmann, Esther Ooi, Dick C. Chan, P. Hugh R. Barrett, Gerald F. Watts, Klaus G. Parhofer
Abstract
In patients with elevated Lp(a), the fractional catabolism of Lp(a) is slower than that of LDL-apoB particles, implying that different metabolic pathways are involved in the catabolism of these lipoproteins. These findings have implications for new therapies for lowering apolipoprotein(a) and apoB to prevent atherosclerotic cardiovascular disease.
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