Cyclin D1‐induced proliferation is independent of beta‐catenin in Head and Neck Cancer
Oral Diseases2013Vol. 20(3), pp. e42–8
Citations Over Time
Katiuchia Uzzun Sales, FS Giudice, RM Castilho, FT Salles, Cristiane H. Squarize, Aline Corrêa Abrahão, Décio dos Santos Pinto Júnior
Abstract
Taken together, we show that the change in cyclin D1 levels is more likely to be due to the EGFR-Akt pathway activation than due to beta-catenin nuclear translocation.
Related Papers
- → Misexpression of Cyclin B3 Leads to Aberrant Spermatogenesis(2006)42 cited
- → Increased cyclin E expression may obviate the role of cyclin D1 during brain development in cyclin D1 knockout mice(2005)16 cited
- → p53‐independent regulation of cyclin B1 in normal human fibroblasts during UV‐induced G2‐arrest(1999)9 cited
- → Effect of wortmannin and 3-AB on G2 phase arrest and transient failure to activate MPF induced by ionizing irradiation(1998)
- Influence on checkpoints and cell apoptosis induced by different dosage of X-ray(2005)