Activation of the Cardiac Proteasome During Pressure Overload Promotes Ventricular Hypertrophy
Circulation2006Vol. 114(17), pp. 1821–1828
Citations Over TimeTop 10% of 2006 papers
Christophe Depré, Qian Wang, Lin Yan, Nadia Hedhli, Pallavi S. Peter, Li Chen, Chull Hong, Luc Hittinger, Bijan Ghaleh, Junichi Sadoshima, Dorothy E. Vatner, Stephen F. Vatner, Kiran Madura
Abstract
The increase in proteasome expression and activity found during chronic pressure overload in myocardium submitted to higher stress is also required for the establishment of LVH.
Related Papers
- → Progressive induction of left ventricular pressure overload in a large animal model elicits myocardial remodeling and a unique matrix signature(2011)62 cited
- → A Minimally Invasive Model of Aortic Stenosis in Swine(2023)1 cited
- Abstract 533: Crucial Role of Rho-Kinase in Pressure Overload--Induced Right Ventricular Hypertrophy and Dysfunction in Mice: A Possible Novel Therapeutic Target of Right Ventricular Failure(2014)
- → Low Left Ventricular Mass in Aortic Stenosis: A Distinct Mode of Adaptation to Pressure Overload Associated with Better Contractility?(2019)
- → Left ventricular hypertrophy induced by aortic constriction in the rat is an unappropriate model to understand pressure overload-induced left ventricular hypertrophy in humans A. van der Laarse, H.W. Vliegen. Department of Cardiology, University Hospital, Leiden, The Netherlands(1988)