The Role of the miR-21/SPRY2 Axis in Modulating Proangiogenic Factors, Epithelial Phenotypes, and Wound Healing in Corneal Epithelial Cells
Investigative Ophthalmology & Visual Science2019Vol. 60(12), pp. 3854–3854
Citations Over TimeTop 10% of 2019 papers
Abstract
TGF-β1 or hypoxia induced miR-21 and inhibited SPRY2, thereby enhancing proangiogenic signaling, suppressing the epithelial phenotype, and promoting wound healing in corneal epithelial cells.
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