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IL-23 Promotes γδT Cell Activity in Dry Eye Disease Progression
Investigative Ophthalmology & Visual Science2025Vol. 66(2), pp. 10–10
Abstract
An increase in conjunctival IL-23 expression contributes to the induction of the DED inflammatory response through interactions with its cognate receptor on γδT cells and the promotion of their proliferation. The findings of this study suggest that the suppression of IL-17A through the blockade of IL-23R activation may be a viable target for improving the management of inflammation in DED patients.
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