Searching for mechanisms that matter in early septic acute kidney injury: an experimental study
Critical Care2011Vol. 15(5), pp. R256–R256
Citations Over TimeTop 10% of 2011 papers
Abstract
The observed variability in susceptibility to septic AKI in our models replicates that of human disease. Early abnormal host response accompanied by subsequent uncoupling between systemic and renal vascular resistance appear to be major determinants in the early phase of porcine septic AKI. Nonuniform and model-related renal hemodynamic responses that are unpredictable from systemic changes should be taken into consideration when evaluating hemodynamic therapeutic interventions in septic AKI.
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