Renal haemodynamic, microcirculatory, metabolic and histopathological responses to peritonitis-induced septic shock in pigs
Critical Care2008Vol. 12(6), pp. R164–R164
Citations Over TimeTop 11% of 2008 papers
J Chvojka, Roman Sýkora, Aleš Kroužecký, Jaroslav Raděj, Veronika Varnerova, Thomas Karvunidis, Ondřej Hes, Ivan Novák, Peter Radermacher, Martin Matějovič
Abstract
The results of this experimental study argue against the concept of renal vasoconstriction and tubular necrosis as physiological and morphological substrates of early septic AKI. Renal venous congestion might be a hidden and clinically unrecognised contributor to the development of kidney dysfunction.
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