Hyperglycemia-Induced Reactive Oxygen Species Toxicity to Endothelial Cells Is Dependent on Paracrine Mediators
Diabetes2008Vol. 57(7), pp. 1952–1965
Citations Over TimeTop 1% of 2008 papers
Abstract
Our in vitro results indicate that HRECs respond to cytokines rather than high glucose, suggesting that in vivo diabetes-related endothelial injury in the retina may be due to glucose-induced cytokine release by other retinal cells and not a direct effect of high glucose.
Related Papers
- → Proinflammatory Cytokines Mediate GPCR Dysfunction(2017)19 cited
- → MicroRNA-345-3p is a potential biomarker and ameliorates rheumatoid arthritis by reducing the release of proinflammatory cytokines(2023)3 cited
- → Induction of inducible nitric oxide synthase and proinflammatory cytokines expression by o,p′-DDT in macrophages(2004)45 cited
- → Dihydrocapsaicin suppresses proinflammatory cytokines expression by enhancing nuclear factor IA in a NF-κB-dependent manner(2016)20 cited
- Effects of Proinflammatory Cytokines in the Heart(2004)