Rolf Stricker
Forschungszentrum Jülich(DE)
Publications by Year
Research Areas
Cellular transport and secretion, Protein Kinase Regulation and GTPase Signaling, Mitochondrial Function and Pathology, Peptidase Inhibition and Analysis, Neuroscience and Neuropharmacology Research
Most-Cited Works
- → Thrombin (PAR-1)-induced proliferation in astrocytes via MAPK involves multiple signaling pathways(2002)217 cited
- → Hetero-oligomerization of the P2Y11 receptor with the P2Y1 receptor controls the internalization and ligand selectivity of the P2Y11 receptor(2007)99 cited
- → Ca2+-dependent permeability transition regulation in rat brain mitochondria by 2′,3′-cyclic nucleotides and 2′,3′-cyclic nucleotide 3′-phosphodiesterase(2009)91 cited
- → Protease‐activated receptor‐1 protects rat astrocytes from apoptotic cell death via JNK‐mediated release of the chemokine GRO/CINC‐1(2006)61 cited
- → The mitochondria permeability transition pore complex in the brain with interacting proteins – promising targets for protection in neurodegenerative diseases(2010)56 cited
- → Calcium-induced permeability transition in rat brain mitochondria is promoted by carbenoxolone through targeting connexin43(2010)52 cited
- → cDNA cloning of porcine p42IP4, a membrane‐associated and cytosolic 42 kDa inositol(1,3,4,5)tetrakisphosphate receptor from pig brain with similarly high affinity for phosphatidylinositol (3,4,5)P3(1997)52 cited
- → In aging, the vulnerability of rat brain mitochondria is enhanced due to reduced level of 2′,3′-cyclic nucleotide-3′-phosphodiesterase (CNP) and subsequently increased permeability transition in brain mitochondria in old animals(2014)46 cited
- → Enzyme-Assisted Total Synthesis of the Optical Antipodes d-myo-Inositol 3,4,5-Trisphosphate and d-myo-Inositol 1,5,6-Trisphosphate: Aspects of Their Structure−Activity Relationship to Biologically Active Inositol Phosphates(1999)43 cited
- → Extracellular α-crystallin protects astrocytes from cell death through activation of MAPK, PI3K/Akt signaling pathway and blockade of ROS release from mitochondria(2015)42 cited