Hua Tang
Qingdao University(CN)Southwest University of Political Science & Law(CN)Hunan Normal University(CN)South China Normal University(CN)Dalian Medical University(CN)University of Florida Health(US)University of Florida(US)Fuyang City People's Hospital(CN)Second Affiliated Hospital of Chongqing Medical University(CN)Shaanxi Provincial Hospital of Traditional Chinese Medicine(CN)Shandong Provincial QianFoShan Hospital(CN)Shandong First Medical University(CN)University of Florida Health Science Center(US)Chongqing Medical University(CN)Florida College(US)Stanford University(US)
Publications by Year
Research Areas
Cancer-related molecular mechanisms research, Hepatitis B Virus Studies, Sirtuins and Resveratrol in Medicine, MicroRNA in disease regulation, Circular RNAs in diseases
Most-Cited Works
- → Programming the magnitude and persistence of antibody responses with innate immunity(2011)963 cited
- → Toll-like receptor–mediated induction of type I interferon in plasmacytoid dendritic cells requires the rapamycin-sensitive PI(3)K-mTOR-p70S6K pathway(2008)386 cited
- → Programming dendritic cells to induce TH2 and tolerogenic responses(2010)345 cited
- → The T helper type 2 response to cysteine proteases requires dendritic cell–basophil cooperation via ROS-mediated signaling(2010)314 cited
- → Amino Acid-Induced Translation of TOP mRNAs Is Fully Dependent on Phosphatidylinositol 3-Kinase-Mediated Signaling, Is Partially Inhibited by Rapamycin, and Is Independent of S6K1 and rpS6 Phosphorylation(2001)298 cited
- → Transduction of Growth or Mitogenic Signals into Translational Activation of TOP mRNAs Is Fully Reliant on the Phosphatidylinositol 3-Kinase-Mediated Pathway but Requires neither S6K1 nor rpS6 Phosphorylation