Ning T. Yeh
National Institutes of Health(US)National Institute on Deafness and Other Communication Disorders(US)
Publications by Year
Research Areas
NF-κB Signaling Pathways, Ubiquitin and proteasome pathways, Peptidase Inhibition and Analysis, Cell death mechanisms and regulation, Angiogenesis and VEGF in Cancer
Most-Cited Works
- Expression of proinflammatory and proangiogenic cytokines in patients with head and neck cancer.(1999)
- Novel proteasome inhibitor PS-341 inhibits activation of nuclear factor-kappa B, cell survival, tumor growth, and angiogenesis in squamous cell carcinoma.(2001)
- Hepatocyte growth factor/scatter factor-induced activation of MEK and PI3K signal pathways contributes to expression of proangiogenic cytokines interleukin-8 and vascular endothelial growth factor in head and neck squamous cell carcinoma.(2001)
- → Effects of pharmacologic antagonists of epidermal growth factor receptor, PI3K and MEK signal kinases on NF‐κB and AP‐1 activation and IL‐8 and VEGF expression in human head and neck squamous cell carcinoma lines(2002)231 cited
- IL (interleukin)-1alpha promotes nuclear factor-kappaB and AP-1-induced IL-8 expression, cell survival, and proliferation in head and neck squamous cell carcinomas.(2001)
- Coexpression of proangiogenic factors IL-8 and VEGF by human head and neck squamous cell carcinoma involves coactivation by MEK-MAPK and IKK-NF-kappaB signal pathways.(2001)
- → A signal network involving coactivated NF‐κB and STAT3 and altered p53 modulates BAX/BCL‐XL expression and promotes cell survival of head and neck squamous cell carcinomas(2008)107 cited
- → A pilot study of longitudinal serum cytokine and angiogenesis factor levels as markers of therapeutic response and survival in patients with head and neck squamous cell carcinoma(2005)98 cited
- → Hepatocyte Growth Factor/Scatter Factor Differentially Regulates Expression of Proangiogenic Factors through Egr-1 in Head and Neck Squamous Cell Carcinoma(2005)80 cited
- → A Novel Nuclear Factor-κB Gene Signature Is Differentially Expressed in Head and Neck Squamous Cell Carcinomas in Association with TP53 Status(2007)75 cited