Paolo Barassi
Windtree Therapeutics (United States)(US)
Publications by Year
Research Areas
Cardiac electrophysiology and arrhythmias, Ion channel regulation and function, Heart Failure Treatment and Management, Ion Transport and Channel Regulation, Receptor Mechanisms and Signaling
Most-Cited Works
- → Organ Hypertrophic Signaling within Caveolae Membrane Subdomains Triggered by Ouabain and Antagonized by PST 2238(2004)149 cited
- → Evidence for an interaction between adducin and Na+-K+-ATPase: relation to genetic hypertension(1999)130 cited
- → Renal Na,K-ATPase in Genetic Hypertension(1996)94 cited
- → Istaroxime stimulates SERCA2a and accelerates calcium cycling in heart failure by relieving phospholamban inhibition(2013)79 cited
- → Istaroxime, a Stimulator of Sarcoplasmic Reticulum Calcium Adenosine Triphosphatase Isoform 2a Activity, as a Novel Therapeutic Approach to Heart Failure(2006)63 cited
- → SERCA2a stimulation by istaroxime improves intracellular Ca2+ handling and diastolic dysfunction in a model of diabetic cardiomyopathy(2021)50 cited
- → Modulation of Sarcoplasmic Reticulum Function by PST2744 [Istaroxime; (E,Z)-3-((2-Aminoethoxy)imino) Androstane-6,17-dione Hydrochloride)] in a Pressure-Overload Heart Failure Model(2008)39 cited
- → 17α-O-(Aminoalkyl)oxime Derivatives of 3β,14β-Dihydroxy-5β-androstane and 3β-Hydroxy-14-oxoseco-D-5β-androstane as Inhibitors of Na+,K+-ATPase at the Digitalis Receptor(2001)31 cited
- → 17β-O-Aminoalkyloximes of 5β-Androstane-3β,14β-diol with Digitalis-like Activity: Synthesis, Cardiotonic Activity, Structure−Activity Relationships, and Molecular Modeling of the Na+,K+-ATPase Receptor(2000)30 cited
- → A New Approach to the Design of Novel Inhibitors of Na+,K+-ATPase: 17α-Substituted Seco-D 5β-Androstane as Cassaine Analogues(1998)29 cited