Joseph Chapo
Gilead Sciences (United States)(US)
Publications by Year
Research Areas
Signaling Pathways in Disease, Histone Deacetylase Inhibitors Research, Nitric Oxide and Endothelin Effects, Cardiac Ischemia and Reperfusion, Phosphodiesterase function and regulation
Most-Cited Works
- → Canonical Transient Receptor Potential Channels Promote Cardiomyocyte Hypertrophy through Activation of Calcineurin Signaling(2006)277 cited
- → Meiotic Recombination Provokes Functional Activation of the p53 Regulatory Network(2010)93 cited
- → A novel kinase inhibitor establishes a predominant role for protein kinase D as a cardiac class IIa histone deacetylase kinase(2009)58 cited
- → Identification of Potent and Selective Amidobipyridyl Inhibitors of Protein Kinase D(2010)41 cited
- → Suppression of HDAC nuclear export and cardiomyocyte hypertrophy by novel irreversible inhibitors of CRM1(2009)30 cited
- → Acute effects of endothelin receptor antagonists on hepatic hemodynamics of cirrhotic and noncirrhotic ratsThis article is one of a selection of papers published in the two-part special issue entitled 20 Years of Endothelin Research.(2010)18 cited
- → Age exacerbates chronic catecholamine-induced impairments in contractile reserve in the rat(2011)7 cited
- → A phosphorimager-based filter binding thyroid hormone receptor competition assay for chemical screening(2006)4 cited
- → Histone deacetylase inhibition (HDACi) attenuates catecholamine‐induced impairments in contractile reserve(2010)
- → Age exacerbates chronic catecholamine‐induced impairments in contractile reserve(2009)