Silvia Sostegni
Friedrich-Alexander-Universität Erlangen-Nürnberg(DE)
Publications by Year
Research Areas
Blood Coagulation and Thrombosis Mechanisms, Ion Channels and Receptors, Coagulation, Bradykinin, Polyphosphates, and Angioedema, Calpain Protease Function and Regulation, Cardiovascular, Neuropeptides, and Oxidative Stress Research
Most-Cited Works
- → Cathepsin S Causes Inflammatory Pain via Biased Agonism of PAR2 and TRPV4(2014)180 cited
- → Neutrophil Elastase Activates Protease-activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 (TRPV4) to Cause Inflammation and Pain(2015)165 cited
- → A TRPA1 antagonist reverts oxaliplatin-induced neuropathic pain(2013)75 cited
- → Biocompatible cationic lipids for the formulation of liposomal DNA vectors(2014)20 cited
- → Sensitisation of TRPV4 by PAR2 is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase(2014)16 cited
- → Cathepsin S causes inflammatory pain via biased agonism of PAR2 and TRPV4.(2014)5 cited
- → Erratum to: Sensitisation of TRPV4 by PAR2 is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase(2017)1 cited
- → 400 Cathepsin S Induces Inflammation and Pain via Biased Agonism of PAR2 and TRPV4(2014)