Nabendu Pore

Publications by Year

Research Areas

Cancer Immunotherapy and Biomarkers, Cytokine Signaling Pathways and Interactions, Lung Cancer Treatments and Mutations, Cancer, Hypoxia, and Metabolism, Tuberculosis Research and Epidemiology

Most-Cited Works

• → Regulation of glut1 mRNA by Hypoxia-inducible Factor-1(2001)740 cited
• Epidermal growth factor receptor transcriptionally up-regulates vascular endothelial growth factor expression in human glioblastoma cells via a pathway involving phosphatidylinositol 3'-kinase and distinct from that induced by hypoxia.(2000)
• → EGFR Tyrosine Kinase Inhibitors Decrease VEGF Expression by Both Hypoxia-Inducible Factor (HIF)-1–Independent and HIF-1–Dependent Mechanisms(2006)263 cited
• → Sp1 Is Involved in Akt-mediated Induction of VEGF Expression through an HIF-1–independent Mechanism(2004)226 cited
• → Akt1 Activation Can Augment Hypoxia-Inducible Factor-1α Expression by Increasing Protein Translation through a Mammalian Target of Rapamycin–Independent Pathway(2006)184 cited
• → Nelfinavir Down-regulates Hypoxia-Inducible Factor 1α and VEGF Expression and Increases Tumor Oxygenation: Implications for Radiotherapy(2006)152 cited
• PTEN mutation and epidermal growth factor receptor activation regulate vascular endothelial growth factor (VEGF) mRNA expression in human glioblastoma cells by transactivating the proximal VEGF promoter.(2003)
• → Phosphatase and Tensin Homologue Deficiency in Glioblastoma Confers Resistance to Radiation and Temozolomide that Is Reversed by the Protease Inhibitor Nelfinavir(2007)128 cited
• → Epidermal Growth Factor Receptor Inhibition Modulates the Microenvironment by Vascular Normalization to Improve Chemotherapy and Radiotherapy Efficacy(2009)123 cited
• → M-CSF Signals through the MAPK/ERK Pathway via Sp1 to Induce VEGF Production and Induces Angiogenesis In Vivo(2008)98 cited