Nabendu Pore
AstraZeneca (United States)(US)
Publications by Year
Research Areas
Cancer Immunotherapy and Biomarkers, Cytokine Signaling Pathways and Interactions, Lung Cancer Treatments and Mutations, Cancer, Hypoxia, and Metabolism, Tuberculosis Research and Epidemiology
Most-Cited Works
- → Regulation of glut1 mRNA by Hypoxia-inducible Factor-1(2001)740 cited
- Epidermal growth factor receptor transcriptionally up-regulates vascular endothelial growth factor expression in human glioblastoma cells via a pathway involving phosphatidylinositol 3'-kinase and distinct from that induced by hypoxia.(2000)
- → EGFR Tyrosine Kinase Inhibitors Decrease VEGF Expression by Both Hypoxia-Inducible Factor (HIF)-1–Independent and HIF-1–Dependent Mechanisms(2006)263 cited
- → Sp1 Is Involved in Akt-mediated Induction of VEGF Expression through an HIF-1–independent Mechanism(2004)226 cited
- → Akt1 Activation Can Augment Hypoxia-Inducible Factor-1α Expression by Increasing Protein Translation through a Mammalian Target of Rapamycin–Independent Pathway(2006)184 cited
- → Nelfinavir Down-regulates Hypoxia-Inducible Factor 1α and VEGF Expression and Increases Tumor Oxygenation: Implications for Radiotherapy(2006)152 cited
- PTEN mutation and epidermal growth factor receptor activation regulate vascular endothelial growth factor (VEGF) mRNA expression in human glioblastoma cells by transactivating the proximal VEGF promoter.(2003)
- → Phosphatase and Tensin Homologue Deficiency in Glioblastoma Confers Resistance to Radiation and Temozolomide that Is Reversed by the Protease Inhibitor Nelfinavir(2007)128 cited
- → Epidermal Growth Factor Receptor Inhibition Modulates the Microenvironment by Vascular Normalization to Improve Chemotherapy and Radiotherapy Efficacy(2009)123 cited
- → M-CSF Signals through the MAPK/ERK Pathway via Sp1 to Induce VEGF Production and Induces Angiogenesis In Vivo(2008)98 cited