Charlotte Bauer
Publications by Year
Research Areas
Alzheimer's disease research and treatments, Nuclear Receptors and Signaling, Mitochondrial Function and Pathology, Prion Diseases and Protein Misfolding, Endoplasmic Reticulum Stress and Disease
Most-Cited Works
- → Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimer’s disease models and human brains(2020)238 cited
- → Intraneuronal aggregation of the β-CTF fragment of APP (C99) induces Aβ-independent lysosomal-autophagic pathology(2016)207 cited
- → The β-Secretase-Derived C-Terminal Fragment of βAPP, C99, But Not Aβ, Is a Key Contributor to Early Intraneuronal Lesions in Triple-Transgenic Mouse Hippocampus(2012)192 cited
- → Post-translational remodeling of ryanodine receptor induces calcium leak leading to Alzheimer’s disease-likepathologies and cognitive deficits(2017)175 cited
- → Localization and Processing of the Amyloid-β Protein Precursor in Mitochondria-Associated Membranes(2016)153 cited
- → MT5-MMP is a new pro-amyloidogenic proteinase that promotes amyloid pathology and cognitive decline in a transgenic mouse model of Alzheimer’s disease(2015)108 cited
- → The transcription factor XBP1s restores hippocampal synaptic plasticity and memory by control of the Kalirin-7 pathway in Alzheimer model(2016)107 cited
- → α-Secretase-derived Fragment of Cellular Prion, N1, Protects against Monomeric and Oligomeric Amyloid β (Aβ)-associated Cell Death(2011)91 cited
- → Genome-wide, high-content siRNA screening identifies the Alzheimer’s genetic risk factor FERMT2 as a major modulator of APP metabolism(2016)78 cited
- → Alzheimer’s genetic risk factor FERMT2 (Kindlin-2) controls axonal growth and synaptic plasticity in an APP-dependent manner(2020)58 cited