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Joseph J. Boyle | doi.page

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Joseph J. Boyle

Canon (Japan)(JP)Queen Mary University of London(GB)Nottinghamshire County Council(GB)Nottinghamshire Healthcare NHS Foundation Trust(GB)Nottingham Biomedical Research Centre(GB)Imperial College London(GB)

Publications by Year

Research Areas

Atherosclerosis and Cardiovascular Diseases, Heme Oxygenase-1 and Carbon Monoxide, Cell Adhesion Molecules Research, Eicosanoids and Hypertension Pharmacology, Complement system in diseases

Most-Cited Works

→ Cells of the adult human heart(2020)1,693 cited
→ Copy number polymorphism in Fcgr3 predisposes to glomerulonephritis in rats and humans(2006)678 cited
→ Coronary Intraplaque Hemorrhage Evokes a Novel Atheroprotective Macrophage Phenotype(2009)309 cited
→ Immunoglobulin M Is Required for Protection Against Atherosclerosis in Low-Density Lipoprotein Receptor–Deficient Mice(2009)247 cited
→ Activating Transcription Factor 1 Directs Mhem Atheroprotective Macrophages Through Coordinated Iron Handling and Foam Cell Protection(2011)212 cited
→ Tumor Necrosis Factor-α Promotes Macrophage-Induced Vascular Smooth Muscle Cell Apoptosis by Direct and Autocrine Mechanisms(2003)192 cited

→ Cooperative Interactions Between RB and p53 Regulate Cell Proliferation, Cell Senescence, and Apoptosis in Human Vascular Smooth Muscle Cells From Atherosclerotic Plaques(1998)191 cited

→ EndoPDI, a Novel Protein-disulfide Isomerase-like Protein That Is Preferentially Expressed in Endothelial Cells Acts as a Stress Survival Factor(2003)176 cited

→ Complement C1q Reduces Early Atherosclerosis in Low-Density Lipoprotein Receptor-Deficient Mice(2007)163 cited

→ Human Blood-Derived Macrophages Induce Apoptosis in Human Plaque-Derived Vascular Smooth Muscle Cells by Fas-Ligand/Fas Interactions(2001)141 cited