Lothar J. Strobl
Helmholtz Zentrum München(DE)
Publications by Year
Research Areas
Viral-associated cancers and disorders, T-cell and B-cell Immunology, Lymphoma Diagnosis and Treatment, Developmental Biology and Gene Regulation, Immune Cell Function and Interaction
Most-Cited Works
- → Loss of intestinal crypt progenitor cells owing to inactivation of both Notch1 and Notch2 is accompanied by derepression of CDK inhibitors p27 Kip1 and p57 Kip2(2008)404 cited
- → Notch2 is required for progression of pancreatic intraepithelial neoplasia and development of pancreatic ductal adenocarcinoma(2010)229 cited
- → Epstein-Barr virus nuclear antigen 2 exerts its transactivating function through interaction with recombination signal binding protein RBP-J kappa, the homologue of Drosophila Suppressor of Hairless.(1994)213 cited
- → Liver‐specific inactivation of Notch2, but not Notch1, compromises intrahepatic bile duct development in mice†‡(2008)210 cited
- → Hold back of RNA polymerase II at the transcription start site mediates down-regulation of c-myc in vivo.(1992)179 cited
- → Hierarchy of Notch–Delta interactions promoting T cell lineage commitment and maturation(2007)177 cited
- → Regulation of monocyte cell fate by blood vessels mediated by Notch signalling(2016)142 cited
- → The Spi-1/PU.1 and Spi-B ets family transcription factors and the recombination signal binding protein RBP-J kappa interact with an Epstein-Barr virus nuclear antigen 2 responsive cis-element.(1994)136 cited
- → RBP-L, a Transcription Factor Related to RBP-Jκ(1997)131 cited
- → EBNA2 and Notch signalling in Epstein–Barr virus mediated immortalization of B lymphocytes(2001)130 cited