Michael Fritsche
Publications by Year
Research Areas
Cancer-related Molecular Pathways, Cancer Research and Treatments, Virus-based gene therapy research, Cytokine Signaling Pathways and Interactions, DNA Repair Mechanisms
Most-Cited Works
- Induction of nuclear accumulation of the tumor-suppressor protein p53 by DNA-damaging agents.(1993)
- → Restoration of the growth suppression function of mutant p53 by a synthetic peptide derived from the p53 C-terminal domain(1997)347 cited
- Flavonoids activate wild-type p53.(1996)
- → Protein interactions at the carboxyl terminus of p53 result in the induction of its in vitro transactivation potential(1997)47 cited
- → Regulation of the trans-activation potential of STAT5 through its DNA-binding activity and interactions with heterologous transcription factors(2000)32 cited
- Enhanced p53 activity and accumulation in response to DNA damage upon DNA transfection.(1995)
- → p53 suppresses cytokine induced, Stat5 mediated activation of transcription(1998)27 cited
- → Nuclear accumulation of p53 in response to treatment with DNA-damaging agents(1994)25 cited
- → Transformation-associated decrease in cell surface binding of neoglycoenzymes in a temperature-sensitive, virally transformed mouse model(1991)5 cited
- → Crosstalk between the extracellular domain of the ErbB2 receptor and IGF-1 receptor signaling(2003)4 cited