Hiroko Omori
The University of Osaka(JP)
Publications by Year
Research Areas
Autophagy in Disease and Therapy, Streptococcal Infections and Treatments, Endoplasmic Reticulum Stress and Disease, Inflammasome and immune disorders, Lysosomal Storage Disorders Research
Most-Cited Works
- → Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1β production(2008)1,986 cited
- → Two Beclin 1-binding proteins, Atg14L and Rubicon, reciprocally regulate autophagy at different stages(2009)1,189 cited
- → The Atg16L Complex Specifies the Site of LC3 Lipidation for Membrane Biogenesis in Autophagy(2008)1,058 cited
- → Atg9a controls dsDNA-driven dynamic translocation of STING and the innate immune response(2009)825 cited
- → Neutrophil Extracellular Traps Mediate a Host Defense Response to Human Immunodeficiency Virus-1(2012)705 cited
- → Autophagy sequesters damaged lysosomes to control lysosomal biogenesis and kidney injury(2013)627 cited
- → An Atg4B Mutant Hampers the Lipidation of LC3 Paralogues and Causes Defects in Autophagosome Closure(2008)504 cited
- → Modulation of Local PtdIns3P Levels by the PI Phosphatase MTMR3 Regulates Constitutive Autophagy(2010)205 cited
- → The LC3 recruitment mechanism is separate from Atg9L1-dependent membrane formation in the autophagic response againstSalmonella(2011)167 cited
- → Two-amino acids change in the nsp4 of SARS coronavirus abolishes viral replication(2017)161 cited