Masayo Shamoto‐Nagai
Aichi Gakuin University(JP)
Publications by Year
Research Areas
Parkinson's Disease Mechanisms and Treatments, Mitochondrial Function and Pathology, Alzheimer's disease research and treatments, Nuclear Receptors and Signaling, Neurotransmitter Receptor Influence on Behavior
Most-Cited Works
- → Mitochondrial Genome Variation in Eastern Asia and the Peopling of Japan(2004)527 cited
- → Type A monoamine oxidase and serotonin are coordinately involved in depressive disorders: from neurotransmitter imbalance to impaired neurogenesis(2017)168 cited
- → Mitochondrial permeability transition mediates apoptosis induced by N‐methyl(R)salsolinol, an endogenous neurotoxin, and is inhibited by Bcl‐2 and rasagiline, N‐propargyl‐1(R)‐aminoindan(2002)165 cited
- → Mitochondria in Neuroprotection by Phytochemicals: Bioactive Polyphenols Modulate Mitochondrial Apoptosis System, Function and Structure(2019)164 cited
- → An inhibitor of mitochondrial complex I, rotenone, inactivates proteasome by oxidative modification and induces aggregation of oxidized proteins in SH‐SY5Y cells(2003)141 cited
- → Oxidative Stress in Mitochondria: Decision to Survival and Death of Neurons in Neurodegenerative Disorders(2005)131 cited
- → An anti-Parkinson's disease drug, N-propargyl-1(R)-aminoindan (rasagiline), enhances expression of anti-apoptotic Bcl-2 in human dopaminergic SH-SY5Y cells(2002)123 cited
- → In parkinsonian substantia nigra, α-synuclein is modified by acrolein, a lipid-peroxidation product, and accumulates in the dopamine neurons with inhibition of proteasome activity(2007)113 cited
- → N-Propargyl-1 (R)-aminoindan, rasagiline, increases glial cell line-derived neurotrophic factor (GDNF) in neuroblastoma SH-SY5Y cells through activation of NF-κB transcription factor(2003)110 cited
- → Selective nitration of mitochondrial complex I by peroxynitrite: involvement in mitochondria dysfunction and cell death of dopaminergic SH-SY5Y cells(2002)102 cited