Oleg Tarnavski
Publications by Year
Research Areas
Cardiac Fibrosis and Remodeling, Congenital heart defects research, Signaling Pathways in Disease, Cardiac electrophysiology and arrhythmias, Cardiomyopathy and Myosin Studies
Most-Cited Works
- → Endothelial-to-mesenchymal transition contributes to cardiac fibrosis(2007)2,170 cited
- → Phosphoinositide 3-kinase(p110α) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy(2003)530 cited
- → Inhibition of mTOR Signaling With Rapamycin Regresses Established Cardiac Hypertrophy Induced by Pressure Overload(2004)519 cited
- → Rapamycin Attenuates Load-Induced Cardiac Hypertrophy in Mice(2003)461 cited
- → The Insulin-like Growth Factor 1 Receptor Induces Physiological Heart Growth via the Phosphoinositide 3-Kinase(p110α) Pathway(2004)397 cited
- → Mouse cardiac surgery: comprehensive techniques for the generation of mouse models of human diseases and their application for genomic studies(2004)318 cited
- → Gata4 is required for maintenance of postnatal cardiac function and protection from pressure overload-induced heart failure(2006)186 cited
- → Deletion of Ribosomal S6 Kinases Does Not Attenuate Pathological, Physiological, or Insulin-Like Growth Factor 1 Receptor-Phosphoinositide 3-Kinase-Induced Cardiac Hypertrophy(2004)122 cited
- Zeisberg, E.M. et al. Endothelial-to-mesenchymal transition contributes to cardiac fibrosis. Nat. Med. 13, 952-961(2007)
- → Alterations in apoptosis regulatory factors during hypertrophy and heart failure(2004)62 cited