Cary Koss
St. Jude Children's Research Hospital(US)
Publications by Year
Research Areas
Acute Myeloid Leukemia Research, Acute Lymphoblastic Leukemia research, Protein Degradation and Inhibitors, Genomic variations and chromosomal abnormalities, Cancer Genomics and Diagnostics
Most-Cited Works
- → An Inv(16)(p13.3q24.3)-Encoded CBFA2T3-GLIS2 Fusion Protein Defines an Aggressive Subtype of Pediatric Acute Megakaryoblastic Leukemia(2012)273 cited
- → Pediatric non–Down syndrome acute megakaryoblastic leukemia is characterized by distinct genomic subsets with varying outcomes(2017)222 cited
- → Proteasome inhibition targets the KMT2A transcriptional complex in acute lymphoblastic leukemia(2023)19 cited
- → Targeted Inhibition of the MLL Transcriptional Complex By Proteosome Inhibitors Elicits a High Response Rate in Relapsed/Refractory MLL Rearranged Leukemia(2014)9 cited
- → KMT2A-Rearranged Infant Acute Lymphoblastic Leukemia Cells Undergo ER-Stress-Induced Apoptosis Following Exposure to Proteasome Inhibitors(2019)1 cited
- → Transcriptome Sequence Analysis of Pediatric Acute Megakaryoblastic Leukemia Identifies An Inv(16)(p13.3;q24.3)-Encoded CBFA2T3-GLIS2 Fusion Protein As a Recurrent Lesion in 39% of Non-Infant Cases: A Report From the St. Jude Children's Research Hospital – Washington University Pediatric Cancer Genome Project(2011)1 cited
- → Identification and Characterization of Novel Fusion Proteins in Pediatric Acute Megakaryoblastic Leukemia(2014)
- → Author Correction: Proteasome inhibition targets the KMT2A transcriptional complex in acute lymphoblastic leukemia(2023)
- → Abstract 4867: Identification of an inv(16)-encoded CBFA2T3-GLIS2 fusion protein in 34% of non-infant acute megkaryoblastic leukemias: A report from the Pediatric Cancer Genome Project(2012)
- → Novel AMKL Chimeric Transcription Factors Are Potent Inducers of Leukemia with Unique Mechanisms of Leukemogenesis(2014)