a public good project by the
Synthesis
Company
of California

© 2026

Jun‐ichiro Inoue | doi.page

0 works0 citations0 h-index

Jun‐ichiro Inoue

Tokyo Medical University(JP)The University of Tokyo(JP)

Publications by Year

Research Areas

NF-κB Signaling Pathways, Immune Response and Inflammation, interferon and immune responses, T-cell and Retrovirus Studies, Cytokine Signaling Pathways and Interactions

Most-Cited Works

→ Induction and Activation of the Transcription Factor NFATc1 (NFAT2) Integrate RANKL Signaling in Terminal Differentiation of Osteoclasts(2002)2,657 cited
→ TAK1 is a ubiquitin-dependent kinase of MKK and IKK(2001)2,091 cited
→ The kinase TAK1 can activate the NIK-IκB as well as the MAP kinase cascade in the IL-1 signalling pathway(1999)1,183 cited
→ Lipopolysaccharide Stimulates the MyD88-Independent Pathway and Results in Activation of IFN-Regulatory Factor 3 and the Expression of a Subset of Lipopolysaccharide-Inducible Genes(2001)1,088 cited
→ Interferon-α induction through Toll-like receptors involves a direct interaction of IRF7 with MyD88 and TRAF6(2004)1,007 cited
→ Severe osteopetrosis, defective interleukin‐1 signalling and lymph node organogenesis in TRAF6‐deficient mice(1999)636 cited

→ Induction of interleukin 2 receptor gene expression by p40x encoded by human T-cell leukemia virus type 1.(1986)512 cited

→ The Tumor Necrosis Factor Family Receptors RANK and CD40 Cooperatively Establish the Thymic Medullary Microenvironment and Self-Tolerance(2008)491 cited

→ Identification of TRAF6, a Novel Tumor Necrosis Factor Receptor-associated Factor Protein That Mediates Signaling from an Amino-terminal Domain of the CD40 Cytoplasmic Region(1996)477 cited

→ Segregation of TRAF6-mediated signaling pathways clarifies its role in osteoclastogenesis(2001)464 cited