Dat P. Ha
University of California, Irvine(US)
Publications by Year
Research Areas
Endoplasmic Reticulum Stress and Disease, Autophagy in Disease and Therapy, Pancreatic function and diabetes, Heat shock proteins research, SARS-CoV-2 and COVID-19 Research
Most-Cited Works
- → The chaperone GRP78 is a host auxiliary factor for SARS-CoV-2 and GRP78 depleting antibody blocks viral entry and infection(2021)168 cited
- → Endoplasmic reticulum stress activates SRC, relocating chaperones to the cell surface where GRP78/CD109 blocks TGF-β signaling(2018)137 cited
- → ER chaperone GRP78/BiP translocates to the nucleus under stress and acts as a transcriptional regulator(2023)120 cited
- → Murine leukemia virus glycosylated Gag blocks apolipoprotein B editing complex 3 and cytosolic sensor access to the reverse transcription complex(2013)93 cited
- → The stress-inducible molecular chaperone GRP78 as potential therapeutic target for coronavirus infection(2020)91 cited
- → The stress-inducible ER chaperone GRP78/BiP is upregulated during SARS-CoV-2 infection and acts as a pro-viral protein(2022)68 cited
- → GRP78 haploinsufficiency suppresses acinar-to-ductal metaplasia, signaling, and mutant Kras -driven pancreatic tumorigenesis in mice(2017)53 cited
- → Suppression of stress induction of the 78-kilodalton glucose regulated protein (GRP78) in cancer by IT-139, an anti-tumor ruthenium small molecule inhibitor(2018)47 cited
- → ER residential chaperone GRP78 unconventionally relocalizes to the cell surface via endosomal transport(2021)40 cited
- → Endoplasmic reticulum chaperone GRP78/BiP is critical for mutant Kras-driven lung tumorigenesis(2021)31 cited