John Nip
Unilever (United States)(US)Data Power Decisions (United States)(US)
Publications by Year
Research Areas
Cell Adhesion Molecules Research, Cancer-related Molecular Pathways, Protease and Inhibitor Mechanisms, Acute Myeloid Leukemia Research, Skin Protection and Aging
Most-Cited Works
- → ETO, a Target of t(8;21) in Acute Leukemia, Makes Distinct Contacts with Multiple Histone Deacetylases and Binds mSin3A through Its Oligomerization Domain(2001)339 cited
- → The t(8;21) fusion protein, AML1–ETO, specifically represses the transcription of the p14ARF tumor suppressor in acute myeloid leukemia(2002)257 cited
- → Human melanoma cells derived from lymphatic metastases use integrin alpha v beta 3 to adhere to lymph node vitronectin.(1992)175 cited
- → Bcl-2 is an apoptotic target suppressed by both c-Myc and E2F-1(2001)166 cited
- → TEL, a Putative Tumor Suppressor, Modulates Cell Growth and Cell Morphology of Ras-Transformed Cells While Repressing the Transcription of stromelysin-1(2000)106 cited
- → Expression of the AML-1 Oncogene Shortens the G1Phase of the Cell Cycle(2000)100 cited
- → E2F-1 Cooperates with Topoisomerase II Inhibition and DNA Damage To Selectively Augment p53-Independent Apoptosis(1997)96 cited
- → Coordinated expression of the vitronectin receptor and the urokinase-type plasminogen activator receptor in metastatic melanoma cells.(1995)83 cited
- → Integrin α3β1 can promote adhesion and spreading of metastatic breast carcinoma cells on the lymph node stroma(1996)66 cited
- Paracrine growth stimulation by hepatocyte-derived insulin-like growth factor-1: a regulatory mechanism for carcinoma cells metastatic to the liver.(1994)