Sally A. Nicholas
Medway School of Pharmacy(GB)University of Kent(GB)
Publications by Year
Research Areas
Cancer, Hypoxia, and Metabolism, Immune Response and Inflammation, Eicosanoids and Hypertension Pharmacology, Cancer-related Molecular Pathways, Inflammasome and immune disorders
Most-Cited Works
- → Deletion of DDB1 in Mouse Brain and Lens Leads to p53-Dependent Elimination of Proliferating Cells(2006)150 cited
- → The involvement of hypoxia-inducible factor 1 alpha in Toll-like receptor 7/8-mediated inflammatory response(2009)63 cited
- → DDB1 is essential for genomic stability in developing epidermis(2007)61 cited
- → Involvement of xanthine oxidase and hypoxia-inducible factor 1 in Toll-like receptor 7/8-mediated activation of caspase 1 and interleukin-1β(2010)56 cited
- → Involvement of hypoxia‐inducible factor‐1 HiF(1α) in IgE‐mediated primary human basophil responses(2009)53 cited
- → The role of redox‐dependent mechanisms in the downregulation of ligand‐induced Toll‐like receptors 7, 8 and 4‐mediated HIF‐1α prolyl hydroxylation(2009)33 cited
- → Hypoxia-inducible Factor 1 as one of the “Signaling Drivers” of Toll-like Receptor-Dependent and Allergic Inflammation(2010)29 cited
- → Dysfunctional mitochondria contain endogenous high-affinity human Toll-like receptor 4 (TLR4) ligands and induce TLR4-mediated inflammatory reactions(2011)19 cited
- → Myeloid cell death associated with Toll-like receptor 7/8-mediated inflammatory response. Implication of ASK1, HIF-1alpha, IL-1beta and TNF-alpha(2010)10 cited
- → Apoptosis signal-regulating kinase 1 (ASK1) and HIF-1α protein are essential factors for nitric oxide-dependent accumulation of p53 in THP-1 human myeloid macrophages(2008)9 cited