Jessica DesAulniers
University of Alberta(CA)Women and Children’s Health Research Institute(CA)
Publications by Year
Research Areas
Chemotherapy-induced cardiotoxicity and mitigation, PI3K/AKT/mTOR signaling in cancer, Cardiac Fibrosis and Remodeling, Iron Metabolism and Disorders, Cardiovascular Function and Risk Factors
Most-Cited Works
- → Angiotensin 1–7 Ameliorates Diabetic Cardiomyopathy and Diastolic Dysfunction in db/db Mice by Reducing Lipotoxicity and Inflammation(2014)187 cited
- → Angiotensin 1–7 mediates renoprotection against diabetic nephropathy by reducing oxidative stress, inflammation, and lipotoxicity(2014)126 cited
- → Iron-overload injury and cardiomyopathy in acquired and genetic models is attenuated by resveratrol therapy(2015)110 cited
- → Females Are Protected From Iron‐Overload Cardiomyopathy Independent of Iron Metabolism: Key Role of Oxidative Stress(2017)52 cited
- → Loss of p47 phox Subunit Enhances Susceptibility to Biomechanical Stress and Heart Failure Because of Dysregulation of Cortactin and Actin Filaments(2013)51 cited
- → Resveratrol mediates therapeutic hepatic effects in acquired and genetic murine models of iron‐overload(2015)50 cited
- → Endothelial and cardiomyocyte PI3Kβ divergently regulate cardiac remodelling in response to ischaemic injury(2018)22 cited
- → PI3Kα is essential for the recovery from Cre/tamoxifen cardiotoxicity and in myocardial insulin signalling but is not required for normal myocardial contractility in the adult heart(2015)19 cited
- → Heterogenous expression of endocrine and progenitor cells within the neonatal porcine pancreatic lobes–Implications for neonatal porcine islet xenotransplantation(2023)3 cited
- Endothelial and Cardiomyocyte PI3Kβ Divergently Regulate Cardiac Remodeling in Response to Ischemic Injury(2018)