Taj D. King
Southern Research Institute(US)United States Department of the Navy(US)
Publications by Year
Research Areas
Wnt/β-catenin signaling in development and cancer, Cancer-related gene regulation, Cell death mechanisms and regulation, Mitochondrial Function and Pathology, Skin and Cellular Biology Research
Most-Cited Works
- → The wnt/β‐catenin signaling pathway: A potential therapeutic target in the treatment of triple negative breast cancer(2011)312 cited
- → Caspase-3 activation induced by inhibition of mitochondrial complex I is facilitated by glycogen synthase kinase-3β and attenuated by lithium(2001)177 cited
- → Frizzled7 as an emerging target for cancer therapy(2011)153 cited
- → AMP-activated protein kinase (AMPK) activating agents cause dephosphorylation of Akt and glycogen synthase kinase-3(2006)137 cited
- → Muscarinic Receptor Activation Protects Cells from Apoptotic Effects of DNA Damage, Oxidative Stress, and Mitochondrial Inhibition(2003)120 cited
- → Silibinin inhibits Wnt/β-catenin signaling by suppressing Wnt co-receptor LRP6 expression in human prostate and breast cancer cells(2012)120 cited
- → Dkk1 Stabilizes Wnt Co-Receptor LRP6: Implication for Wnt Ligand-Induced LRP6 Down-Regulation(2010)75 cited
- → Inhibition of glycogen synthase kinase-3 protects cells from intrinsic but not extrinsic oxidative stress(2005)66 cited
- → Unregulated mitochondrial GSK3β activity results in NADH:Ubiquinone oxidoreductase deficiency(2008)55 cited
- → The protein phosphatase-1/inhibitor-2 complex differentially regulates GSK3 dephosphorylation and increases sarcoplasmic/endoplasmic reticulum calcium ATPase 2 levels(2006)25 cited