Lisa Salazar
University of California, Irvine(US)
Publications by Year
Research Areas
Genetic Neurodegenerative Diseases, Pluripotent Stem Cells Research, Protein Tyrosine Phosphatases, Fibroblast Growth Factor Research, Mitochondrial Function and Pathology
Most-Cited Works
- → Potential function for the Huntingtin protein as a scaffold for selective autophagy(2014)296 cited
- → Developmental alterations in Huntington's disease neural cells and pharmacological rescue in cells and mice(2017)230 cited
- → Receptor Tyrosine Kinases Activate Canonical WNT/β-Catenin Signaling via MAP Kinase/LRP6 Pathway and Direct β-Catenin Phosphorylation(2012)164 cited
- → Analysis of STAT1 Activation by Six FGFR3 Mutants Associated with Skeletal Dysplasia Undermines Dominant Role of STAT1 in FGFR3 Signaling in Cartilage(2008)47 cited
- → Bisindolylmaleimide I Suppresses Fibroblast Growth Factor-mediated Activation of Erk MAP Kinase in Chondrocytes by Preventing Shp2 Association with the Frs2 and Gab1 Adaptor Proteins(2006)36 cited
- → STAT1 and STAT3 do not participate in FGF-mediated growth arrest in chondrocytes(2008)35 cited
- → Fibroblast Growth Factor Receptor 3 Interacts with and Activates TGFβ-Activated Kinase 1 Tyrosine Phosphorylation and NFκB Signaling in Multiple Myeloma and Bladder Cancer(2014)33 cited
- → Huntingtin contains an ubiquitin-binding domain and regulates lysosomal targeting of mitochondrial and RNA-binding proteins(2024)8 cited
- → Prevention of Transgene Silencing During Human Pluripotent Stem Cell Differentiation(2025)6 cited