Julie Bastien
Novartis (Switzerland)(CH)Novartis Institutes for BioMedical Research
Publications by Year
Research Areas
Retinoids in leukemia and cellular processes, Estrogen and related hormone effects, Ubiquitin and proteasome pathways, Protein Kinase Regulation and GTPase Signaling, Mitochondrial Function and Pathology
Most-Cited Works
- → Nuclear retinoid receptors and the transcription of retinoid-target genes(2004)766 cited
- → Selective VPS34 inhibitor blocks autophagy and uncovers a role for NCOA4 in ferritin degradation and iron homeostasis in vivo(2014)724 cited
- → Involvement of MINK, a Ste20 Family Kinase, in Ras Oncogene-Induced Growth Arrest in Human Ovarian Surface Epithelial Cells(2005)105 cited
- → TFIIH Interacts with the Retinoic Acid Receptor γ and Phosphorylates Its AF-1-activating Domain through cdk7(2000)103 cited
- → Single-cell and bulk transcriptomics of the liver reveals potential targets of NASH with fibrosis(2021)92 cited
- → A Role for p38 Stress-Activated Protein Kinase in Regulation of Cell Growth via TORC1(2009)89 cited
- → Phosphorylation of the Retinoid X Receptor at the Omega loop, modulates the expression of retinoic-acid-target genes with a promoter context specificity(2005)52 cited
- → Down-regulation of the Phosphatidylinositol 3-Kinase/Akt Pathway Is Involved in Retinoic Acid-induced Phosphorylation, Degradation, and Transcriptional Activity of Retinoic Acid Receptor γ2(2002)51 cited
- → Preferential amplification of a human mitochondrial DNA deletion in vitro and in vivo(2018)44 cited
- → The Phosphorylation Site Located in the A Region of Retinoic X Receptor α Is Required for the Antiproliferative Effect of Retinoic Acid (RA) and the Activation of RA Target Genes in F9 Cells(2002)42 cited