John S. Bett
University of Dundee(GB)MRC Protein Phosphorylation and Ubiquitylation Unit(GB)University of Glasgow(GB)
Publications by Year
Research Areas
Ubiquitin and proteasome pathways, Genetic Neurodegenerative Diseases, Mitochondrial Function and Pathology, Endoplasmic Reticulum Stress and Disease, Autophagy in Disease and Therapy
Most-Cited Works
- → UBQLN2 Mediates Autophagy-Independent Protein Aggregate Clearance by the Proteasome(2016)305 cited
- → Suppression of protein aggregation by chaperone modification of high molecular weight complexes(2012)113 cited
- → Proteasome impairment does not contribute to pathogenesis in R6/2 Huntington's disease mice: exclusion of proteasome activator REGγ as a therapeutic target(2005)102 cited
- → Proteostasis regulation by the ubiquitin system(2016)71 cited
- → Molecular chaperones and photoreceptor function(2008)68 cited
- → The Ubiquitin-Proteasome Reporter GFPu Does Not Accumulate in Neurons of the R6/2 Transgenic Mouse Model of Huntington's Disease(2009)60 cited
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