Mami Oki
Hiroshima University(JP)
Research Areas
Endoplasmic Reticulum Stress and Disease, Pancreatic function and diabetes, NF-κB Signaling Pathways, interferon and immune responses, Galectins and Cancer Biology
Most-Cited Works
- → Unfolded protein response, activated by OASIS family transcription factors, promotes astrocyte differentiation(2012)79 cited
- → The Endoplasmic Reticulum Stress Transducer OASIS Is involved in the Terminal Differentiation of Goblet Cells in the Large Intestine(2012)72 cited
- → The Endoplasmic Reticulum Stress Transducer BBF2H7 Suppresses Apoptosis by Activating the ATF5-MCL1 Pathway in Growth Plate Cartilage(2012)38 cited