B. Obermaier–Kusser
Klinikum Ludwigshafen(DE)
Publications by Year
Research Areas
Mitochondrial Function and Pathology, Metabolism and Genetic Disorders, ATP Synthase and ATPases Research, Metabolism, Diabetes, and Cancer, Protein Kinase Regulation and GTPase Signaling
Most-Cited Works
- → A defective Intramolecular Autoactivation Cascade May Cause the Reduced Kinase Activity of the Skeletal Muscle Insulin Receptor from Patients with Non-insulin-dependent Diabetes Mellitus(1989)109 cited
- → Prevention by Protein Kinase C Inhibitors of Glucose-Induced Insulin-Receptor Tyrosine Kinase Resistance in Rat Fat Cells(1991)98 cited
- → Distinct .alpha.-subunit structures of human insulin receptor A and B variants determine differences in tyrosine kinase activities(1992)80 cited
- → Different mechanisms inferred from sequences of human mitochondrial DNA deletions in ocular myopathies(1991)71 cited
- → Altered pattern of insulin receptor isotypes in skeletal muscle membranes of Type 2 (non-insulin-dependent) diabetic subjects(1993)66 cited
- → Mapping of heteroplasmic mitochondrial DNA deletions in Kearns-Sayre syndrome(1989)66 cited
- → Mitochondrial myopathies: divergences of genetic deletions, biochemical defects and the clinical syndromes(1990)58 cited
- → Further evidence for a two-step model of glucose-transport regulation. Inositol phosphate-oligosaccharides regulate glucose-carrier activity(1989)51 cited
- → Neurological disorders in members of families with Leber's hereditary optic neuropathy (LHON) caused by different mitochondrial mutations(1995)45 cited
- → An altered IGF-I receptor is present in human leukemic cells.(1990)44 cited