Margarete Hafley
The University of Texas MD Anderson Cancer Center(US)
Publications by Year
Research Areas
Galectins and Cancer Biology, Glycosylation and Glycoproteins Research, Wnt/β-catenin signaling in development and cancer, Peptidase Inhibition and Analysis, Signaling Pathways in Disease
Most-Cited Works
- → Overexpressed Galectin-3 in Pancreatic Cancer Induces Cell Proliferation and Invasion by Binding Ras and Activating Ras Signaling(2012)128 cited
- → Galectin-3 Mediates Tumor Cell–Stroma Interactions by Activating Pancreatic Stellate Cells to Produce Cytokines via Integrin Signaling(2017)106 cited
- → Cell-surface galectin-3 confers resistance to TRAIL by impeding trafficking of death receptors in metastatic colon adenocarcinoma cells(2011)98 cited
- → Low expression of galectin-3 is associated with poor survival in node-positive breast cancers and mesenchymal phenotype in breast cancer stem cells(2016)42 cited
- → Cell surface galectin-3 defines a subset of chemoresistant gastrointestinal tumor-initiating cancer cells with heightened stem cell characteristics(2016)25 cited
- → Mucin 21 confers resistance to apoptosis in an O-glycosylation-dependent manner(2022)18 cited
- → Activation of Vitamin D/VDR Signaling Reverses Gemcitabine Resistance of Pancreatic Cancer Cells Through Inhibition of MUC1 Expression(2023)15 cited
- → Dysregulated KLF4 expression plays a pivotal role in the pathogenesis of pancreatic intraductal papillary mucinous neoplasms(2024)5 cited
- → Abstract 4328: Galectin-3 mediates tumor-stromal interaction by activating pancreatic stellate cell through integrinβ1/ILK/NF-κB signaling(2017)1 cited
- → Su1728 A TRAIL-Resistant Subset of Colon Cancer Initiating Stem Cells Characterized by Surface Galectin-3(2013)