Mary Jo Wenning
Indiana University IndianapolisIndiana University – Purdue University Indianapolis(US)
Publications by Year
Research Areas
Neurofibromatosis and Schwannoma Cases, Nitric Oxide and Endothelin Effects, Hemoglobinopathies and Related Disorders, Neuroendocrine Tumor Research Advances, Genomics, phytochemicals, and oxidative stress
Most-Cited Works
- → Neurofibromin-deficient Schwann cells secrete a potent migratory stimulus for Nf1+/– mast cells(2003)212 cited
- → Genetic and Biochemical Evidence That Haploinsufficiency of the Nf1 Tumor Suppressor Gene Modulates Melanocyte and Mast Cell Fates in Vivo(2000)183 cited
- → Hyperactivation of P21ras and the Hematopoietic-Specific Rho Gtpase, Rac2, Cooperate to Alter the Proliferation of Neurofibromin-Deficient Mast Cells in Vivo and in Vitro(2001)135 cited
- → Lymphoproliferative defects in mice lacking the expression of neurofibromin: functional and biochemical consequences ofNf1 deficiency in T-cell development and function(2002)52 cited
- → Functional p85α gene is required for normal murine fetal erythropoiesis(2003)46 cited
- → K-Ras is essential for normal fetal liver erythropoiesis(2005)31 cited
- → Leukemic potential of doubly mutant Nf1 andWv hematopoietic cells(2003)15 cited
- → Neurofibromatosis Type 1 Gene Haploinsufficiency Reduces AP-1 Gene Expression without Abrogating the Anabolic Effect of Parathyroid Hormone(2006)9 cited
- → Redox signaling inhibition reverses the age‐related impairment in shear‐mediated collateral growth(2013)