Ke Ding
Nanjing University of Information Science and Technology(CN)Nanjing University(CN)
Publications by Year
Research Areas
Autophagy in Disease and Therapy, Traumatic Brain Injury and Neurovascular Disturbances, Heme Oxygenase-1 and Carbon Monoxide, Genomics, phytochemicals, and oxidative stress, Neuroinflammation and Neurodegeneration Mechanisms
Most-Cited Works
- → Melatonin stimulates antioxidant enzymes and reduces oxidative stress in experimental traumatic brain injury: the Nrf2–ARE signaling pathway as a potential mechanism(2014)226 cited
- → Fucoxanthin provides neuroprotection in models of traumatic brain injury via the Nrf2-ARE and Nrf2-autophagy pathways(2017)192 cited
- → Luteolin provides neuroprotection in models of traumatic brain injury via the Nrf2–ARE pathway(2014)191 cited
- → Inhibition of cathepsin S induces autophagy and apoptosis in human glioblastoma cell lines through ROS-mediated PI3K/AKT/mTOR/p70S6K and JNK signaling pathways(2014)155 cited
- → Melatonin protects the brain from apoptosis by enhancement of autophagy after traumatic brain injury in mice(2015)98 cited
- → Melatonin reduced microglial activation and alleviated neuroinflammation induced neuron degeneration in experimental traumatic brain injury: Possible involvement of mTOR pathway(2014)98 cited
- → Necrostatin-1 Ameliorates Intracerebral Hemorrhage-Induced Brain Injury in Mice Through Inhibiting RIP1/RIP3 Pathway(2015)90 cited
- → FTY720 induces autophagy-related apoptosis and necroptosis in human glioblastoma cells(2015)87 cited
- → Traumatic Brain Injury-Induced Neuronal Apoptosis is Reduced Through Modulation of PI3K and Autophagy Pathways in Mouse by FTY720(2015)85 cited
- → Mollugin induces tumor cell apoptosis and autophagy via the PI3K/AKT/mTOR/p70S6K and ERK signaling pathways(2014)79 cited