Gangwen Han
Peking University(CN)Peking University International Hospital(CN)
Publications by Year
Research Areas
TGF-β signaling in diseases, PARP inhibition in cancer therapy, Kruppel-like factors research, Wound Healing and Treatments, Hedgehog Signaling Pathway Studies
Most-Cited Works
- → Caspase 3–mediated stimulation of tumor cell repopulation during cancer radiotherapy(2011)887 cited
- → Smad4 loss in mice causes spontaneous head and neck cancer with increased genomic instability and inflammation(2009)217 cited
- → Role of TGFβ-Mediated Inflammation in Cutaneous Wound Healing(2006)216 cited
- → Targeted Disruption of TGF-β/Smad3 Signaling Modulates Skin Fibrosis in a Mouse Model of Scleroderma(2004)212 cited
- → Keratinocyte-specific Smad2 ablation results in increased epithelial-mesenchymal transition during skin cancer formation and progression(2008)208 cited
- → MicroRNA-31 Is Overexpressed in Psoriasis and Modulates Inflammatory Cytokine and Chemokine Production in Keratinocytes via Targeting Serine/Threonine Kinase 40(2012)207 cited
- → Genome-Wide Expression Profiling of Five Mouse Models Identifies Similarities and Differences with Human Psoriasis(2011)202 cited
- → Smad7-Induced β-Catenin Degradation Alters Epidermal Appendage Development(2006)163 cited
- → The Pro-inflammatory Role of TGFβ1: A Paradox?(2012)153 cited
- → IKKα is a critical coregulator of a Smad4-independent TGFβ-Smad2/3 signaling pathway that controls keratinocyte differentiation(2008)148 cited