Anastasia Galvita
Institute of Theoretical and Experimental Biophysics(RU)Otto-von-Guericke University Magdeburg(DE)
Publications by Year
Research Areas
Mitochondrial Function and Pathology, Neuroscience and Neuropharmacology Research, Adenosine and Purinergic Signaling, Cholinesterase and Neurodegenerative Diseases, Phosphodiesterase function and regulation
Most-Cited Works
- → Ca2+-dependent permeability transition regulation in rat brain mitochondria by 2′,3′-cyclic nucleotides and 2′,3′-cyclic nucleotide 3′-phosphodiesterase(2009)91 cited
- → In aging, the vulnerability of rat brain mitochondria is enhanced due to reduced level of 2′,3′-cyclic nucleotide-3′-phosphodiesterase (CNP) and subsequently increased permeability transition in brain mitochondria in old animals(2014)46 cited
- → The brain‐specific protein, p42IP4(ADAP 1) is localized in mitochondria and involved in regulation of mitochondrial Ca2+(2009)22 cited
- → Identification of phosphorylated form of 2′, 3′-cyclic nucleotide 3′-phosphodiesterase (CNPase) as 46 kDa phosphoprotein in brain non-synaptic mitochondria overloaded by calcium(2014)17 cited
- → Novel protein partners in the control of the brain mitochondrial permeability transition: 2’,3’-cyclic nucleotide 3’-phosphodiesterase (CNP) and p42IP4(centaurin α1; ADAP 1)(2010)
- → The neuron-specific protein, p42IP4 (Centaurin-α1) is localized in mitochondria, interacts with 2',3'-cyclic nucleotide 3'-phosphodiesterase and is involved in regulation and control of mitochondrial Ca2+(2009)
- → In brain mitochondria, calcium and cell death-associated permeability transition are controlled by possibly associated proteins, 2',3'-CNPase, Centaurin-alpha1 and peripheral benzodiazepine receptor, and their substrates/ligands(2009)