Junji Kamon
Nissan (United Kingdom)(GB)Nissan Chemical Corporation (Japan)(JP)
Publications by Year
Research Areas
Adipokines, Inflammation, and Metabolic Diseases, Adipose Tissue and Metabolism, Peroxisome Proliferator-Activated Receptors, Metabolism, Diabetes, and Cancer, Regulation of Appetite and Obesity
Most-Cited Works
- → The fat-derived hormone adiponectin reverses insulin resistance associated with both lipoatrophy and obesity(2001)4,904 cited
- → Adiponectin stimulates glucose utilization and fatty-acid oxidation by activating AMP-activated protein kinase(2002)4,196 cited
- → Cloning of adiponectin receptors that mediate antidiabetic metabolic effects(2003)3,155 cited
- → Targeted disruption of AdipoR1 and AdipoR2 causes abrogation of adiponectin binding and metabolic actions(2007)1,380 cited
- → Impaired Multimerization of Human Adiponectin Mutants Associated with Diabetes(2003)980 cited
- → Globular Adiponectin Protected ob/ob Mice from Diabetes and ApoE-deficient Mice from Atherosclerosis(2003)913 cited
- → The Mechanisms by Which Both Heterozygous Peroxisome Proliferator-activated Receptor γ (PPARγ) Deficiency and PPARγ Agonist Improve Insulin Resistance(2001)636 cited
- → Insulin/Foxo1 Pathway Regulates Expression Levels of Adiponectin Receptors and Adiponectin Sensitivity(2004)544 cited
- → Generation of Globular Fragment of Adiponectin by Leukocyte Elastase Secreted by Monocytic Cell Line THP-1(2004)318 cited
- → Inhibition of RXR and PPARγ ameliorates diet-induced obesity and type 2 diabetes(2001)292 cited