a public good project by the
Synthesis
Company
of California

© 2026

Yuzo Takada | doi.page

0 works0 citations0 h-index

Yuzo Takada

Nippon Veterinary and Life Science University(JP)Japan Fisheries Research and Education Agency(JP)

Publications by Year

Research Areas

Gout, Hyperuricemia, Uric Acid, Alcohol Consumption and Health Effects, Liver Disease Diagnosis and Treatment, Case Reports on Hematomas, Forensic and Genetic Research

Most-Cited Works

→ Decreased extra-renal urate excretion is a common cause of hyperuricemia(2012)677 cited
→ Common Defects of ABCG2, a High-Capacity Urate Exporter, Cause Gout: A Function-Based Genetic Analysis in a Japanese Population(2009)430 cited
→ Genome-wide association study of clinically defined gout identifies multiple risk loci and its association with clinical subtypes(2015)182 cited
→ ABCG2 dysfunction causes hyperuricemia due to both renal urate underexcretion and renal urate overload(2014)159 cited
→ Common dysfunctional variants in ABCG2 are a major cause of early-onset gout(2013)131 cited
→ Common dysfunctional variants of ABCG2 have stronger impact on hyperuricemia progression than typical environmental risk factors(2014)94 cited

→ Subtype-specific gout susceptibility loci and enrichment of selection pressure on ABCG2 and ALDH2 identified by subtype genome-wide meta-analyses of clinically defined gout patients(2020)

61 cited

→ A common missense variant of monocarboxylate transporter 9 (MCT9/SLC16A9) gene is associated with renal overload gout, but not with all gout susceptibility(2013)57 cited

→ ABCG2 Dysfunction Increases the Risk of Renal Overload Hyperuricemia(2014)42 cited

→ A common variant of leucine-rich repeat-containing 16A (LRRC16A) gene is associated with gout susceptibility(2013)34 cited