Ashraf Elamin
Philip Morris International (Switzerland)(CH)
Publications by Year
Research Areas
Air Quality and Health Impacts, Chronic Obstructive Pulmonary Disease (COPD) Research, Smoking Behavior and Cessation, Advanced Proteomics Techniques and Applications, Carcinogens and Genotoxicity Assessment
Most-Cited Works
- → Toxicity of the main electronic cigarette components, propylene glycol, glycerin, and nicotine, in Sprague-Dawley rats in a 90-day OECD inhalation study complemented by molecular endpoints(2017)132 cited
- → An 8-Month Systems Toxicology Inhalation/Cessation Study in Apoe−/−Mice to Investigate Cardiovascular and Respiratory Exposure Effects of a Candidate Modified Risk Tobacco Product, THS 2.2, Compared With Conventional Cigarettes(2015)105 cited
- → A 7-month cigarette smoke inhalation study in C57BL/6 mice demonstrates reduced lung inflammation and emphysema following smoking cessation or aerosol exposure from a prototypic modified risk tobacco product(2015)103 cited
- → Alterations in the sputum proteome and transcriptome in smokers and early-stage COPD subjects(2015)86 cited
- → Proteomics for systems toxicology(2014)60 cited
- → A systems toxicology approach for comparative assessment: Biological impact of an aerosol from a candidate modified-risk tobacco product and cigarette smoke on human organotypic bronchial epithelial cultures(2016)59 cited
- → Systems Toxicology Assessment of the Biological Impact of a Candidate Modified Risk Tobacco Product on Human Organotypic Oral Epithelial Cultures(2016)58 cited
- → Effects of Cigarette Smoke, Cessation, and Switching to Two Heat-Not-Burn Tobacco Products on Lung Lipid Metabolism inC57BL/6andApoe−/−Mice—An Integrative Systems Toxicology Analysis(2015)57 cited
- → Evaluation of the Tobacco Heating System 2.2. Part 7: Systems toxicological assessment of a mentholated version revealed reduced cellular and molecular exposure effects compared with mentholated and non-mentholated cigarette smoke(2016)55 cited
- → A six-month systems toxicology inhalation/cessation study in ApoE−/− mice to investigate cardiovascular and respiratory exposure effects of modified risk tobacco products, CHTP 1.2 and THS 2.2, compared with conventional cigarettes(2019)53 cited